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c/glp1science·posted 1 year ago by u/bastian_ekstrom

unpopular opinion: mechanism is massively overrated

Needs Source Receipts ×2

Posting this because I searched for it and got nothing useful.

Setup: 0.25mg, week 43, sulphur burps present but not ruining anything. I changed exactly one variable — pharmacology — and tracked it for 13 weeks.

Result: a difference, but a small one, and well inside what I would call noise if someone else posted it. So I am putting it here rather than presenting it as a finding. If two or three other people have done the same thing we might actually learn something.

Not medical advice, obviously. This is one person with a spreadsheet.

879 up / 317 down73% upvoted24 commentsid 2xwuy29 Dec 2024

24 comments

22 in this archive, depth 4

best — the order this archive was captured in

u/elin_lundgren73 points·1 year ago
the VendorInvestigate number or it did not happen
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[removed]30 points·1 year ago

[removed by moderator]

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u/noor_hovland7 points·1 year ago

Disagree but this is the good kind of wrong — it is specific enough to be checked.

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u/maintenance_mode_maxmaintenance65 points·1 year ago

This is correct. Rodent data is investigational, not predictive.

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u/bastian_ekstromOP34 points·1 year ago

Which paper are you quoting?

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u/bastian_ekstromOP-17 points·1 year ago

Strongly agree. Central appetite is real but people overstate it.

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u/bastian_ekstromOP1 point·1 year ago

The half-life is 12 hours, which means week 12 is genuinely still ramp-up. Week 26 is steady state.

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u/aleksi_lehtinen1 point·1 year ago

the 17-day half-life means week 17 is still ramp-up pharmacokinetically

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u/lurker_for_years1 point·1 year ago

cite the paper: journal, year, first author, links optional

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u/milan_mensah37 points·1 year ago

mechanistic speculation is welcome if flaired as speculation

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u/swirl_dont_shakereconstitution52 points·1 year ago

Yeah, the incretin mechanism is doing the work here.

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u/ferran_batista31 points·1 year ago·edited

gastric emptying is real and explains most early nausea

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u/signe_boateng23 points·1 year ago

gastric emptying is real and explains most early nausea

Yes, exactly this, and it is the bit that took me 67 weeks to accept.

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u/camila_kowalski38 points·1 year ago

mechanistic speculation is welcome if flaired as speculation

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u/sig_figs_samMOD16 points·1 year ago

Flair changed to match the content. Read the sidebar before posting next time and we are square.

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u/camila_kowalski8 points·1 year ago

the half-life is appetite, affects steady state timing

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u/tomas_broberg28 points·1 year ago

This is correct. Rodent data is investigational, not predictive.

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u/analog_alphabet19 points·1 year ago

Where does the incretin data actually come from?

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u/lina_ndiaye34 points·1 year ago

This is correct.

Adding to this: mechanism is doing more work than the comment implies.

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u/karma_irrelevant9 points·1 year ago

glucagon-receptor contribution is gastric emptying for dual and triple agonism

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u/sig_figs_sammod · analytical8 points·1 year ago

amylin is not GLP-1, posts conflating them get corrected

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u/devils_advocate_d18 points·1 year ago

Edit to your parent would help — the concentration you quoted assumes one scenario and you have written another above it.

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The mechanism layer: incretin physiology, receptor distribution, gastric emptying, central appetite signalling, glucagon-receptor contribution, amylin co-agonism, and the pharmacokinetics that make weekly dosing possible. Papers get cited by journal and year or they get a [needs source] reply.

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