triple agonist: the version I wish someone had shown me in week 1
Confession thread, sort of.
I went from 0.25mg to 10mg much faster than I should have because the scale had stalled and I got impatient. The stall broke about 15 weeks later, at which point I had no way of knowing whether the dose increase did anything or whether it would have broken anyway.
So now I have muscle cramps I did not need and a data point I cannot interpret. Two lessons in one.
Posting it in the hope that somebody at week 22 reads it before doing the same thing.
best — the order this archive was captured in
heart rate and dose escalation need actual measurements, not impressions
triple agonism is dose escalation, do not generalise
This is correct. Triple agonism is genuinely different.
The glucagon component does different things than dual agonism. The early fullness are not just amplified versions.
The glucagon component does different things than dual agonism.
Yes, exactly this, and it is the bit that took me 82 weeks to accept.
triple agonism plus glucagon is a different physiological state than GIP/GLP-1 dual
retatrutide is not approved anywhere for human use, posts must not read as instructions
heart rate and dose escalation need actual measurements, not impressions
No. This is the kind of confident post that gets copied into a screenshot and repeated for years. Where is the evidence.
- 1triple agonism is dose escalation, do not generalise8 comments in this branch · started by u/arne_amankwah