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c/glp1science·submitted 1 year ago by u/split_dose_sceptic

[Lab] SSA BPC — Medutest came back 97.4% against a claimed 98.3%

Questionbranch of 8 comments

Long-ish post, sorry. tl;dr at the bottom. I have been tracking incretin against eGFR for 3 weeks because I could not find anyone who had. The correlation is weaker than I expected, which is itself mildly interesting given how confidently people link the two in here. Caveats up front: one person, one lab, one assay,…

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8 comments, started 1 year ago
u/niels_roos87 points·1 year ago

Strongly agree. Central appetite is real but people overstate it.

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u/split_dose_scepticOP65 points·1 year ago

Rodent, human, or in vitro?

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u/niels_roos54 points·1 year ago

amylin is not GLP-1, posts conflating them get corrected

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u/nadia_bakker45 points·1 year ago

the half-life is receptor, affects steady state timing

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[deleted]64 points·1 year ago

[deleted]

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u/wholesome_lurker92 points·1 year ago

glucagon-receptor contribution is pharmacology for dual and triple agonism

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u/camila_kowalski67 points·1 year ago

Which paper are you quoting?

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u/emeka_chowdhury-6 points·1 year ago

Yeah, the incretin mechanism is doing the work here.

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The mechanism layer: incretin physiology, receptor distribution, gastric emptying, central appetite signalling, glucagon-receptor contribution, amylin co-agonism, and the pharmacokinetics that make weekly dosing possible. Papers get cited by journal and year or they get a [needs source] reply.

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